As such, future progress is likely to involve multivariate analyses that compare the characteristics (directional dominance, effect size, allelic spectrum) of CVs that affect multiple traits in the same or opposite directions with respect to fitness. In this article we have given an abbreviated overview of the conceptual and methodological bases

of research at the intersection of evolutionary psychology and behavioral genetics, as well as a sample of the findings in this still nascent field. We have mentioned contributions of evolutionary behavioral genetics to our understanding of mate preferences, sexual dimorphism, sexual maturation, reproductive success, personality, and schizophrenia, this website but of necessity omitted important research on other

traits 58, 59, 60, 61, 62, 63 and 64•]. We have tried to convey some of the depth and breadth of the possibilities afforded by these approaches and hope that this might spur others to adopt these approaches in testing hypotheses in evolutionary psychology and behavioral genetics. Nothing declared. Papers of particular interest, published within the period of review, have been highlighted as: • of CDK inhibitor special interest The authors thank Dr Patrick Sullivan for sharing the CNV effects that are included in Figure 1. This work was supported by National Institutes of Mental Health grants K01MH085812 and R01MH100141 to Dr Keller and an Australian Research Council Discovery Early Career Research Award (DE120100562) to Dr Zietsch. “
“Current Opinion in Behavioral Sciences 2015, 2:81–88 This review comes from a themed issue on Behavioral

genetics unless 2015 Edited by William Davies and Laramie Duncan http://dx.doi.org/10.1016/j.cobeha.2014.10.001 2352-1546/© 2014 The Authors. Published by Elsevier Ltd. This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/3.0/). This review covers quantitative genetic literature on psychotic experiences (PEs) over the last four years (2011–2014). ‘PEs’ are used here to refer to normal traits in the general population, such as paranoia (see also schizotypal traits for more personality-based constructs), that at the extreme are characteristic of symptoms of psychotic disorders such as schizophrenia [1]. Quantitative genetic research aims to investigate the genetic and environmental influences on quantitative phenotypes [2]. PEs are common [3] and are associated with many negative consequences, including increased risk of suicide 4 and 5]. Furthermore, PEs are risk factors for schizophrenia, a potentially debilitating illness and one of the UK’s most resource-consuming brain disorders [6]. As such, research on PEs can not only help us understand PEs themselves, but may also shed light on the neurodevelopment that underlies psychotic illness.